{"_buckets": {"deposit": "ae2c7230-0c77-4f71-8dfc-ae053f6574dc"}, "_deposit": {"created_by": 3, "id": "77", "owners": [3], "pid": {"revision_id": 0, "type": "depid", "value": "77"}, "status": "published"}, "_oai": {"id": "oai:kwmed.repo.nii.ac.jp:00000077", "sets": ["332"]}, "author_link": ["127415", "127416", "127417", "127418"], "item_3_biblio_info_12": {"attribute_name": "書誌情報", "attribute_value_mlt": [{"bibliographicIssueDates": {"bibliographicIssueDate": "1983", "bibliographicIssueDateType": "Issued"}, "bibliographicIssueNumber": "2", "bibliographicPageEnd": "80", "bibliographicPageStart": "73", "bibliographicVolumeNumber": "9", "bibliographic_titles": [{}, {"bibliographic_title": "Kawasaki medical journal", "bibliographic_titleLang": "en"}]}]}, "item_3_description_15": {"attribute_name": "表示順", "attribute_value_mlt": [{"subitem_description": "5", "subitem_description_type": "Other"}]}, "item_3_description_16": {"attribute_name": "アクセション番号", "attribute_value_mlt": [{"subitem_description": "KJ00010227479", "subitem_description_type": "Other"}]}, "item_3_description_9": {"attribute_name": "記事種別(英)", "attribute_value_mlt": [{"subitem_description": "Original Article", "subitem_description_type": "Other"}]}, "item_3_identifier_14": {"attribute_name": "URL", "attribute_value_mlt": [{"subitem_identifier_type": "URI", "subitem_identifier_uri": "http://igakkai.kms-igakkai.com/wp/wp-content/uploads/1983en/9(2)73-80.1983.pdf | http://igakkai.kms-igakkai.com/wp/wp-content/uploads/1983en/9(2)73-80.1983.pdf"}]}, "item_3_relation_20": {"attribute_name": "DOI", "attribute_value_mlt": [{"subitem_relation_type_id": {"subitem_relation_type_id_text": "https://doi.org/10.11482/KMJ-E9(2)73 ", "subitem_relation_type_select": "DOI"}}]}, "item_3_source_id_1": {"attribute_name": "雑誌書誌ID", "attribute_value_mlt": [{"subitem_source_identifier": "AA00710169", "subitem_source_identifier_type": "NCID"}, {"subitem_source_identifier": "AA12029005", "subitem_source_identifier_type": "NCID"}]}, "item_3_source_id_19": {"attribute_name": "ISSN", "attribute_value_mlt": [{"subitem_source_identifier": "0385-0234", "subitem_source_identifier_type": "ISSN"}, {"subitem_source_identifier": "2434-3404", "subitem_source_identifier_type": "ISSN"}]}, "item_3_text_3": {"attribute_name": "論文名よみ", "attribute_value_mlt": [{"subitem_text_value": "Development of Changes in VEP and Associate Neuropathology on Creutzfeldt-Jakob Disease"}]}, "item_3_text_7": {"attribute_name": "著者所属(英)", "attribute_value_mlt": [{"subitem_text_language": "en", "subitem_text_value": "Department of Ophthalmology, Kawasaki Medical School"}, {"subitem_text_language": "en", "subitem_text_value": "Department of Ophthalmology, Kawasaki Medical School"}, {"subitem_text_language": "en", "subitem_text_value": "Division of Neurology, Department of Medicine, Kawasaki Medical School"}, {"subitem_text_language": "en", "subitem_text_value": "Division of Human Pathology (Neuropathology), Department of Pathology, Kawasaki Medical School"}]}, "item_3_textarea_11": {"attribute_name": "抄録(英)", "attribute_value_mlt": [{"subitem_textarea_language": "en", "subitem_textarea_value": "A case, 74-year-old female, of Creutzfeldt-Jakob disease (CJD) was reported with a chronological changes of visual evoked potentials (VEPs) and neuropathological findings. The disease started with a disturbance of visual integration and developed to blindness, myoclonus, mental deterioration and akinetic mutism. A clinical diagnosis was made by a periodic synchronous discharge of EEG and other neurological specificity. Autopsy findings showed a peculiar spongy degeneration of the cerebral cortex. The flash VEP showed a loss of w-shaped wave and a marked delay of the peak latencies in the early stage. Subsequently, the once delayed latency was shortened with the advance of the illness in the middle stage and the N70-P100 amplitude became a huge triphasic wave like inversed ERG. Topographic distribution of the focus of huge component appeared on the left parieto-occipital region. At the terminal stage, the amplitude of huge component reduced. The mechanism of specific VEP changes in this case was interpreted to be due to the loss of generator due to spongy degeneration and the existence of cell fusion in the occipital cortex. 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